APRR9 Antibody

1. PRR9, a core circadian component, is a key regulator of leaf senescence via positive regulation of ORE1 through a feed-forward pathway involving posttranscriptional regulation by miR164 and direct transcriptional regulation. PMID: 30065116
2. Transcriptional co-regulators PRR9, PRR7 and PRR5 inhibit morning loops LHY and CCA1 expression by binding to their promoters. PMID: 24267177
3. PPR9 regulates the promoter activity of LWD1/2. PMID: 21357491
4. Results reveal a role of PRR7 and PRR9 in regulating CCA1 and LHY activities in response to ambient temperature. PMID: 21098730
5. PRR9, PRR7, and PRR5 proteins are major transcriptional repressors of CCA1 and LHY and are thus essential for proper clock function. PMID: 20233950
6. single prr7-3 or prr9-1 mutants exhibit modest period lengthening, but the prr7-3 prr9-1 double mutant shows dramatic and more than additive period lengthening in the light and becomes arrhythmic in constant darkness PMID: 15705949
7. The APRR9 promoter contains at least two distinctive and separable regulatory cis-elements: an "L element" responsible for the light-induced expression, followed by an "R element" necessary for the fundamental rhythmic expression of the gene. PMID: 15725665
8. PRR9/PRR7/PRR5 together act as period-controlling factors, and they play overlapping and distinctive roles close to (or within) the central oscillator in which the relative, PRR1/TOC1, plays an essential role PMID: 15767265
9. The role of clock-associated genes PRR9, PRR7 and PRR5 are involved in activation of CONSTANS and CO-FLOWERING LOCUS T are reported. PMID: 17504813
10. Data show that PRR9 polypeptides oscillate diurnally, and that they accumulate rapidly in response to light, and further suggest that the presence of PRR9 polypeptides is controlled through proteasome-mediated programmed degradation in the dark. PMID: 17890242

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KEGG: ath:AT2G46790

STRING: 3702.AT2G46790.1

UniGene: At.1675